Orthographic working memory deficits in dysgraphia arise primarily from lesions of the left parietal cortex centered on the intraparietal sulcus¶

Claim · All claims
Evidence · 1 study · associational r1 · q2
Currency · newest study 2016 · not yet checked against its sources · not reviewed by a person · overdue for a recheck since 2021: baselines and conditions change

Subclaims¶

q2 i? Lesion mapping localized orthographic working memory deficits primarily to left parietal cortex centered on the intraparietal sulcus. → Rapp 2016

Evidence¶

Rapp 2016¶

Rapp, Brenda; Purcell, Jeremy; Hillis, Argye; Capasso, Rita; Miceli, Gabriele. (2016). Neural bases of orthographic long-term memory and working memory in dysgraphia. BRAIN. https://doi.org/10.1093/brain/awv348

q2 · i? · associational · r1

Voxel-based lesion-mapping analysis of the same post-stroke dysgraphia sample, with participants classified on behavioral profiles alone. The article reports "orthographic working memory deficits primarily arising from lesions of the left parietal cortex centered on the intraparietal sulcus." No effect size is printed.

"orthographic working memory deficits primarily arising from lesions of the left parietal cortex centered on the intraparietal sulcus"

Discussion¶

Related Claims¶

• Orthographic long-term memory deficits in dysgraphia are centered in the left posterior inferior frontal region or left ventral temporal cortex
• Behavioral profiles alone can identify dysgraphia cases with deficits selectively affecting orthographic long-term memory or working memory
• Lesion evidence links written-language processes to language, working memory, and other cognitive skills
• Distinct neural substrates selectively support orthographic long-term memory and orthographic working memory in spelling — a broader claim this one bears on